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Therefore, therapeutic agents capable of mitigating oxidative damage are of significant interest

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Bibliography Robbins, Zachary

In a chronic restraint stress (CRS) model, ER stress is activated through the PERK pathway, upregulating phosphorylated eukaryotic initiation factor 2 (P-eIF2) and CCAAT/enhancer binding protein (C/EBP) homologous protein expression, leading to damage of the jejunal and colonic mucosa and disruption of barrier function [85]

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