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In agreement with a role of HMOX1 as a driver of immunosuppression, myeloid-specific HMOX1 deletion restored CD8 + T cell function and enhanced the therapeutic efficacy of antitumor vaccines [80]

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They also help the liver work better, which has a big part to play in making glutathione

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HT was first described in families with symptoms suggestive of MC mediator release in association with increased TPSAB1 copy numbers