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Here we report that these two oncogenic pathways may not act in concert, but act apparently partially antagonistic at least in human GIC models, potentially accounting for disappointing results with inhibition of either pathway in the clinic 29,30,31 , but also providing new concepts on how to modulate these pathways more efficiently to improve the outcome of glioblastoma

Kidney stones: Overdose of niacin increases the risk of kidney stones

Testing of therapies in a novel nebulin nemaline myopathy model demonstrate a lack of efficacy