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Mitochondrial and synaptic abnormalities in ASD-related syndromes The growing body of evidence reveals that the ability of mitochondria to adapt to the changing environment and energy demand is impaired in various ASD-related syndromes [325, 326]
However, most published series remain small and histologically heterogeneous, limiting insight into subtype-specific radiosensitization
doi:10.5056/jnm.2013.19.1.18
To evaluate the target-binding capacity of D-CuP, molecular docking simulations were conducted using AutoDock Vina against the functional domains of p53 (DNA-binding domain) and HDAC7 (catalytic deacetylase domain)
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