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Diamond LE, et al
Mining these data reveals that many known regulators of GLUT4 traffic harbour insulin-regulated phosphosites that are dysregulated in insulin resistance (Table 1)
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These findings highlight GSHs therapeutic potential in managing chronic diseases by addressing oxidative stress and cellular dysfunction (11)
Key points Amyotrophic lateral sclerosis (ALS), with a lifetime risk of ~1/350, represents an area of huge unmet need and is a useful model of neurodegeneration, with measurable changes in motor function over a relatively short time frame
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