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Zhang et al., 2025a

MGL has received particular attention in recent years due to the discovery that the enzyme is responsible for the inactivation of 2-arachidonoylglycerol (2-AG) which is an endogenous cannabinoid (endocannabinoid)

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no clinical trial data exist for this pairing, though their complementary amylin and incretin-based mechanisms provide a theoretical scientific rationale

You can listen to the full episode here this discussion begins right at 0:02 How it fits into a broader protocol Brecka does not position glutathione as a standalone solution