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These neurotrophic effects correlate with structural neuronal changes observed in animal models, where PSYs have induced increases in dendritic spine density and complexity, including the upregulation of specific synaptic proteins like PSD95 and GAP43 [93], even after a single dose, a phenomenon termed psychoplastogenic effects [48,51]
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Following HGF-mediated dimerization and autophosphorylation of the c-Met receptor, signaling proteins are recruited to the carboxy-terminal docking site, either directly or indirectly through growth factor receptor-bound protein 2 (Grb2) and Grb2-associated binder-1 (Gab1)
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