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In summary, we identified ER stress as the main adaptive stress response caused by AQ-QI and DEAQ-QI and evaluated the protective role of GSTP1 using a transfected HepG2 cell model

From a neurobiological standpoint, the development and intensification of migraine into a debilitating condition involve intricate interactions between neurons, glial cells, vascular components, and inflammatory mediators within the TVS (6, 912)

However, there are also polysynaptic short latency connections with critical structures, such as the basal ganglia [28, 29], in addition to other poorly defined, but likely functionally very important connections to the hypothalamus [30] and hippocampus [31]

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J.MuntnerP.AlonsoA.BittencourtM