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Within this pathological network, mtROS and NLRP3 inflammasome activation form a critical molecular hub: mitochondrial dysfunction induces complexes I and III of the electron transport chain to generate large amounts of mtROS, which triggers NLRP3 inflammasome activation through pathways such as promoting the binding of TXNIP to NLRP3, releasing oxidized mitochondrial DNA (ox-mtDNA) as a co-activator, and modulating mitochondrial membrane potential

doi: 10.1074/jbc.M109.067207

53 FriederichM.HansellP.PalmF

"Cuando un medicamento est en escasez, los que lo componen pueden ser capaces de preparar una versin del medicamento si se cumplen ciertos requisitos federales", dice Amberly Johnson, PharmD, toxiclogo clnico y director del Centro de Control de Envenenamientos de Utah

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