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The mechanistic core of the finding was that Dihexa activated the PI3K/AKT signaling pathway in the brains of the APP/PS1 mice, and that the PI3K inhibitor wortmannin reversed Dihexas anti-inflammatory and anti-apoptotic effects [5]

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HPLC validation performed per batch

Access this chapter Subscribe and save Buy Now Tax calculation will be finalised at checkout Purchases are for personal use only Similar content being viewed by others References Abe M, Wu Z, Yamamoto M, Jin JJ, Tabara Y, Mogi M, Kohara K, Miki T, Nakura J (2005) Association of dopamine -hydroxylase polymorphism with hypertension through interaction with fasting plasma glucose in Japanese