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The primary mechanism of action investigated in ex vivo and in vivo models involves the modulation of the nitric oxide (NO) system and the promotion of angiogenesis

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[3] So the two molecules embody two strategies that converged on the same goal from opposite engineering philosophies: cagrilintide adds a separate hormone (amylin) intended to be combined with an incretin, while retatrutide packs an additional receptor activity (glucagon) into the incretin molecule itself