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A key mechanism underlying this synergy involves alcohol-induced activation of LPLA2 via ATF4-dependent transcription, thereby elevating bis(monoacylglycero)phosphate (BMP) levels in tumors

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doi: 10.1007/s00011-010-0165-9 113

Our data reveal that Rho pathway activation enhances F-actin polymerization, which decreases G-actin-dependent p53 nuclear translocation and transcriptional activity, especially in the setting of genomic instability caused by IR stress

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