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Chronic GH secretion suppresses the anti-lipolytic action of insulin and increases FFA flux into the systemic circulation, thereby promoting lipotoxicity, which causes pathophysiological problems, including insulin resistance [10]

Enhanced accumulation of Kir4.1 protein, but not mRNA, in a murine model of cuprizone-induced demyelination

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Furthermore, liposomes can degrade, destabilising their lipid bilayer structure, which results in the premature release of the encapsulated drug