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Studies have shown that A1AT has the potential to induce tolerance in pre-clinical models of GVHD, particularly gastrointestinal steroid-refractory seems to be an optimal setting
While various risk factors for enhanced APAP toxicity have emerged in the numerous experimental models of steatosis, including induction of Cyp2E1, reduced phase II metabolism, mitochondrial dysfunction, etc., opposite effects have also been described, suggesting that steatosis may not affect toxicity [180]
J Indust Manag Optimiz 10(4) Ethics declarations Conflict of interest The authors declare no competing interests
[DOI] [PMC free article] [PubMed] [Google Scholar] 22.Stahl S.M
By contrast, ranidine therapy led to even more increased portal pressure values than those present in control animals
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