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While direct comparison is complicated by different trial designs and durations, the available data allows reasonable estimates

This review summarizes glucose metabolism in the normal heart and highlights the features and regulatory mechanisms of glucose metabolic reprogramming in myocarditis, including the hypoxia-inducible factor-1/mammalian target of rapamycin axis, nuclear factor erythroid 2-related factor 2-mediated pentose phosphate pathway, immune-responsive gene 1/itaconate axis, and phosphoglycerate kinase 1