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We theorize that the reason dendritic cells have anti-tumor effects, despite that they are professional phagocytes, is because the less destructive microenvironment of the dendritic cells phagosomes is not suitable for the pathogens, as these pathogens have evolved to survive in more extreme microenvironments akin to the phagosomes of macrophages and neutrophils

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The previously determined ability of acrolein to bind to and activate the TRPA1 channel and elicit algesic responses may be a mechanism of the phenomenon seen in this study